CCL5 was found to be remarkably elevated in tumor tissues collected from PTC patients coexistent with HT than those without HT [48], and CXCL9, CXCL10, CXCL11, and CXCL13 were also reportedly upregulated in thyroid tissues from HT patients [26,49]. This evidence concerns the gene CXCL11 and neoplasm.