Moreover, stimulating pancreatic tumor cells with UTP or suramin, an agonist of the nucleotide receptor P2Y2, activated cell proliferation by a pathway implicating CaMK-II and c-Src, likely using mechanisms similar to the ones described above for the CaMK-II/c-Src/MAPK pathway in breast cancer cells [88]. The gene discussed is SRC; the disease is breast carcinoma.