Thus, the identification of a FOXL2 variant must lead to reconsider the diagnosis as granulosa cell tumor with SCTAT-like features, which is one of the main diagnostic pitfalls (Figure 10M,N) [173], especially since colocalizations of SCTAT (with STK11 variant) and granulosa cell tumors (with FOXL2 variant) have been reported [174]. This evidence concerns the gene FOXL2 and granulosa cell tumor.