This study began with successive extractions of de-oiled seeds and rinds (Figure 1), and each extract was focused on anti-AD-associated factors, including DPPH radical-scavenging activities, AChE-inhibitory activities, anti-Aβ1-42 peptide aggregations, and neuroprotective functions against H2O2-induced and Aβ25-35 peptide-induced neuronal cell death in vitro (Figure 2). This evidence concerns the gene ACHE and Alzheimer disease.