MST1 and familial dilated cardiomyopathy: Although the specifics of how CDR1as modulates MST1 ubiquitination remain unexplored in the study, it suggests an indirect interaction with MST1 and possible direct interactions with regulatory factors affecting the ubiquitination of MST1 in the process, thus exerting downstream effects on the Hippo-signaling pathway involved in the pathogenesis of DCM.