To examine whether Axl signaling was required for wound repair, we intraperitoneally injected mice 3 hr prior to injury with either a control IgG Ab or anti-Axl function blocking Ab, which binds to Axl’s extracellular domain and blocks Axl-mediated viral infection (Retallack et al., 2016), and has been shown to inhibit Axl activity in vitro (Bauer et al., 2012; Figure 5A). Here, AXL is linked to viral infectious disease.