Given that previous studies have already pointed out that therapeutic FMT controlled intestinal inflammation through IL-10 secretion by immune cells and microbiota-derived SCFAs promoted T helper 1 (Th1) cell IL-10 production to maintain intestinal homeostasis (29), such similarity may imply that a more robust suppression of the inflammatory response may be the critical factor for the anti-colitis effect of GA and aminated GA in which microbiota can be a significant contributor. Here, IL10 is linked to colitis.