METTL14 and breast cancer: Regarding the regulation of m6A writers, it has been shown that LNC942 straightforwardly attracts METTL14 by carrying particular recognition sequences, thereby maintaining the expression of LNC942 downstream targets in vitro and in vivo through post-transcriptional m6A methylation modifications, which exerted a robust oncogenic effect in promoting breast cancer cell growth and development of colonies along with inhibiting apoptosis potent oncogenic effects (Sun et al., 2020b).