KIF3C and posterior cortical atrophy: Among non-coding RNAs, the METTL3 is imperative for DGCR8 to regulate pri-miRNAs in PCa, where experiments have revealed that m6A modification-dependent METTL3 can interact with DGCR8 to enhance the recognition of prior-miR-182 in PCa, thereby promoting the maturation of pri-miRNAs, leading to PCa proliferation, in addition, to mediate m6A modification of KIF3C mRNA, thereby promoting PCa progression [170, 171].