The latter's research were also perfectly correlated with another recent one indicating that a blockade of TIM3 could not only increase antitumor CD8 T cells activation and IFN‐γ secretion, but would also favour cDC1 cells and CD8 T cells to colocalize within the tumour, amplifying the chance to mount an appropriate antitumor response both in time and space (Gardner et al., 2022). This evidence concerns the gene HAVCR2 and neoplasm.