Moreover, our study also discovered that LTE overtly inhibited the indicators related to inflammation, such as inflammatory cell numbers (neutrophils) in lung, which were produced by neutrophils; besides, LTE significantly declined IL-6, IL-1β, TNF-α, and IL-10 levels in BALF of ALI mice and LPS-evoked BEAS-2B cells. The gene discussed is TNF; the disease is acute respiratory distress syndrome.