Additionally, there have already been several researches uncovering the intermediating effects of hnRNPK between lncRNAs and β-catenin, and in detail, lncRNA CASC11 interacted with hnRNPK and then activated Wnt/β-catenin signaling to augment the growth and metastasis in colorectal cancer [49]. This evidence concerns the gene HNRNPK and colorectal cancer.