LEP and interstitial lung disease: In our study, within the differential alveolar profile, we identified several inflammatory mediators with well-known roles in chemotaxis as GRO/CXCL1 or TARC/CCL17, regulators of matrix remodeling as TGF-β and metalloproteinases regulators as TIMP-2 and other mediators as leptin or IGFBP-3 that are not commonly associated with ILD.