Of these, IL-8/CXCL8, MCP-1/CCL2 and MIG/CXCL9 were identified as significantly increased in IPF versus PLCH and SR-ILD, whereas TARC/CCL17, leptin, and IP-10/CXCL10 showed significant increases in PLCH group (Table 2). This evidence concerns the gene CXCL8 and idiopathic pulmonary fibrosis.