Taken together, these data illustrate that at the 5 days post stroke, ischemic stroke-mediated proinflammatory damage of ChP is subsided and that there is simultaneous restorative activation of the CD11b+CD45+ cells/CD206+ population myeloid cells for tissue repair, which is consistent with previous reports (Rayasam et al., 2020). The gene discussed is PTPRC; the disease is stroke disorder.