APOH and metabolic dysfunction-associated steatotic liver disease: Next, KEGG analysis showed that APOH-associated genes were enriched in the “nonalcoholic fatty liver disease” pathway for the GSE167523 dataset (Fig. 7E), whereas genes from the GSE162694 dataset were mainly enriched in “protein processing in endoplasmic reticulum,” “oxidative phosphorylation,” “peroxisome,” and “proteasome” pathways (Fig. 7F).