Although cardiac neutrophil counts were unaffected in Lcn2−/− bone marrow chimeras after MI, ventricular arrhythmia burden was reduced in Lcn2−/− bone marrow chimeras compared with Lcn2+/+ controls supporting the idea that Lcn2 contributes to arrhythmia burden in the ischemic myocardium (9). This evidence concerns the gene LCN2 and Ventricular arrhythmia.