PPARA and neoplasm: Receptor‐interacting protein kinase 3 (RIPK3) is a central factor in necroptosis, and hence, RIPK3 deficiency not only decreases reactive oxygen species but also significantly inhibits caspase1‐mediated PPAR cleavage, promoting fatty acid metabolism containing fatty acid oxidation (FAO), inducing M2 polarization in the tumor microenvironment.99