TRPC6 and Duchenne muscular dystrophy: Suppression of the pathological mediator of hypertrophy and fibrosis TRPC6 (transient receptor potential cation channel, subfamily C, member 6), using BI749327 has shown efficacy in reducing cardiac fibrosis and increasing muscle function, abating the pro‐fibrotic response in the DMD mice and significantly increasing survival (Lin et al., 2022).