DMD and Duchenne muscular dystrophy: The varied genetic aetiology of DMD complicates the use of patient‐specific genetic corrections, and unfortunately full‐length dystrophin is too large to be packaged into current vectors for direct delivery (Pichavant et al., 2011), but shorter, spliced functional dystrophin has been shown to prevent fibrosis and increase muscle function in mdx mice (Potter et al., 2021).