Interestingly, the supernatants’ ability to promote monocyte migration towards MCP-1 (Figure 5B) as well as macrophage-to-foam cell transition (Figure 5C), two critical mechanisms driving the atherosclerosis process (Ilhan and Kalkanli, 2015; Boring et al., 1998; Tacke et al., 2007), were significantly inhibited by Calprotectin blockade. The gene discussed is CCL2; the disease is atherosclerosis.