NKAP and gastric cancer: found that exosomal circPRRX1 interfered with GC cell proliferation, migration, invasion, and radiosensitivity by MTT, cell colony formation, and transwell assays, and that mechanistically, exosomal circPRRX1 acted as a ceRNA for miR-596 to upregulate NF-κB activating protein (NKAP) thereby regulating GC development and radiosensitivity (90).