,76 IL-17A seems to stimulate epithelial-to-mesenchymal transition, TGF-β secretion, and TGFβ receptor expression at the site of inflammation, a finding that has been mainly demonstrated in liver fibrosis.76, 77, 78 IL-17A stimulates fibroblasts to release granulocyte-monocyte colony stimulating factor (GM-CSF) leading to an influx of monocytes which in turn release IL-6, TGF-β, and IL-23 (Central Illustration C, Table 1).76 Here, TGFB1 is linked to Hepatic fibrosis.