LMNA and amyloidosis: Some of the other interesting candidate proteins are prelamin-A/C (LMNA) (mutations: G602S and R624H) and CHCHD10 (mutation: P34S), which have been shown to aggregate when mutated (24, 25), and KRT74 (mutation: D482N), since other keratin proteins are amyloidogenic (26–29) and the KRT74 mutation is associated with a hypotrichosis of the scalp (30), which is a disorder that is sometimes associated with the amyloidosis of another protein called corneodesmosin (31).