Consistent with the intracellular production levels of IFN-γ in CD4 T cells, the concentration of IFN-γ in the culture medium of CD4- and FimH-stimulated CD11b+CD103- DCs was considerably upregulated compared to that in PBS-treated CD11b+CD103- DCs from DSS-colitis mice (Figure 7C). This evidence concerns the gene CD4 and colitis.