According to the post-mortem examination following the 62-day long AOM/DSS model, the number of visible intestinal polyps and their sizes in M-ILK KO -deficient mice was reduced significantly relative to control mice, suggesting that myeloid-specific ILK loss suppresses colitis-associated colon tumorigenesis (Figures 1B-E). The gene discussed is ILK; the disease is intestinal polyp.