Pregnancy may inhibit the pro-inflammatory T-helper1 cell response, which may mask tuberculosis symptoms while increasing susceptibility to new infections and tuberculosis reactivation [8]. Several alterations in cellular immunity occur during the final stage of pregnancy, including an increase in phagocyte number and activity, plasmoid dendritic cells, and a decrease in natural killer cell cytotoxicity and interferon-gamma production. This evidence concerns the gene IFNG and tuberculosis.