High levels of IL-4, IFN-ɣ, and IL-17 were determined in the P–C group, as corroborated by researchers in lupus conditions, who reported that high-level expression of IFN-ɣ, IL-17 in lupus-like disease could contribute to the tissue damage [132–135]. This evidence concerns the gene IFNA1 and systemic lupus erythematosus.