BAP1 and Parkinson disease: BAP1 overexpression or miR-150-5p inhibition mitigate sh-NEAT1-mediated inhibition of ferroptosis.41 Together, these observations indicate that upregulation of NEAT1 promotes MPP+-induced ferroptosis by regulating the miR-150-5p/BAP1/SLC7A11 pathway in SK-N-SH cells.41 miR-335 was found to promote ferroptosis through the degradation of FTH1 in vivo and in vitro in 6-hydroxydopamine (6-OHDA)-induced models of PD.263 Midbrain dopamine oxidation promotes ferroptosis in dopaminergic neurons by facilitating NEDD4-mediated ubiquitination of GPX4.240