STING1 and Parkinson disease: In accordance with this finding, Szegoet al. [2] also demonstrated that the density of dopaminergic axon terminals (fibers) and the concentration of dopamine in the striatum was lower inSTING knock-in (ki) mice than in STING wild-type (WT) mice, while the concentration of dopamine metabolites was higher inSTING ki mice, suggesting STING contributes to the loss of dopaminergic neurons in Parkinson’s disease (PD).