CLEC10A and cancer: However, in light of the assumptions of the hu-FEDS hypothesis and the interactions described for MGL suggesting that cancer cells and some pathogens may exploit the immunoregulatory function of MGL as part of a survival strategy, the presence of MGL-recognized glycans on fetal tissues, amniotic fluid, placenta, and seminal plasma strongly suggests the involvement of this lectin in immunosuppression and the development of maternal immunological tolerance towards foreign antigens.