In a different study, Nam et al. [101] further indicated that 39 significantly blocked tyrosyl phosphorylation of Stat5, while inhibiting Stat5 DNA-binding activity in human K562, KCL-22M, and primary CML cell lines, with treatments with 39 strongly decreasing the autophosphorylation of Src and SFKs in K562 and KCL-22M cells at 5 μM, and in primary CML cells. This evidence concerns the gene SRC and chronic myelogenous leukemia, BCR-ABL1 positive.