The selection of the molecular target, namely, dual-specificity tyrosine kinase 2 (DYRK2), is based on the numerous data pointing towards its ability to orchestrate a variety of cellular responses, linking genomic stability, tumor cell growth, tumor cell apoptosis, tumor angiogenesis, and tumor cell metabolism as well [66,67,68,69,70]. Here, DYRK2 is linked to neoplasm.