Previous studies have shown that AhR activation by potent AhR agonists such as 6-Formylindolo [3,2-b] carbazole (FICZ) and β-naphloflavone (βNF) improves intestinal permeability and tight junction integrity in in vitro and mouse models of intestinal inflammation [24,26,32]. The gene discussed is AHR; the disease is gastroenteritis.