Also, the levels of other transcripts, such as PPAR-γ, lipoprotein lipase (LPL), adiponectin, and leptin in fetal perirenal fat have been associated with obesity in sheep [24], suggesting that maternal obesity may increase the lipogenic capacity of adipose tissue, increasing the lipid storage and hence the adiposity in the offspring [13]. Here, LEP is linked to obesity due to melanocortin 4 receptor deficiency.