ACTA1 and laryngotracheoesophageal cleft: We observed a large decrease in the expression of several characteristic fibrosis genes [16,26], including Collagen1a1, Collagen3, α-SMA, TGF-β, CTGF, and TIMP1, in the LC group, demonstrating that the administration of Li05 effectively mitigated inflammation in the liver and suppressed the fibrotic process triggered by inflammation.