We observed a large decrease in the expression of several characteristic fibrosis genes [16,26], including Collagen1a1, Collagen3, α-SMA, TGF-β, CTGF, and TIMP1, in the LC group, demonstrating that the administration of Li05 effectively mitigated inflammation in the liver and suppressed the fibrotic process triggered by inflammation. This evidence concerns the gene CCN2 and laryngotracheoesophageal cleft.