CXCR4 and glioblastoma: Over-expression of KLF9 suppressed glioblastoma cell stemness, in part, by repressing transcription of members of multiple signaling pathways that promote oncogenesis and stem cell phenotype, i.e., integrin, CXCR4 (C-X-C Chemokine Receptor Type 4), and Notch pathways [193].