The activation of Akt is known to block amplification of caspase activity via inactivating proapoptotic molecules [84,85,86] Previously, we reported that the increased activation of caspase-3 correlates with increased apoptosis and decreased NSCLC cell viability [20,35,36,37,38,40,41,42]. Here, CASP3 is linked to non-small cell lung carcinoma.