We therefore infer that the alternative pathway is mainly involved in the inflammatory response causing heterophil infiltration after AE17 infection; the Complement and coagulation cascades pathway also showed high levels of FGA, FGB, and FGG differential ploidy; as a fibronectin class, it can be enhanced in this process by binding to interleukins (IL), thereby increasing resistance to infection [25]. This evidence concerns the gene FN1 and infection.