CAMK2G and post-traumatic stress disorder: In this regard, our findings not only corroborate the notion that reactivated memory processing dynamics are more complex than originally thought, suggesting the possibility that extinction memory is open to editing during recall, but also indicate that, through its interaction with GluN2B-NMDAR, CaMKII regulates the competition between antagonistic mnemonic representations and may be a suitable pharmacological target to treat PTSD.