These included neutrophils from healthy control individuals and those with mild to moderate or severe COVID-19 (Combes et al., dataset 1)40, human neutrophils stimulated with G-CSF, IFN-β or IFN-γ (Montaldo et al., datasets 2+3)41 and mouse neutrophils infected with E. coli (Xie et al., dataset 4)7. This evidence concerns the gene IFNB1 and COVID-19.