Ni and team found that inhibiting the lncRNA ZFAS1 attenuates diabetic cardiomyopathy (DCM) progression via sponging of miR-150-5p to activate cyclin D2 (CCND2), which leads to reduced collagen deposition and inhibiting of cardiomyocyte apoptosis and ferroptosis. Here, CCND2 is linked to diabetic cardiomyopathy.