The reduction of serum amyloid P-component, a protein with well-known regulatory functions in inflammation, is in line with the restoration of the gastric mucosa after H. pylori eradication.51 Beta-hexosaminidase subunit beta, a marker of degranulation triggered by H. pylori with a role in bacterial pathogen clearance, and protein S100-A6, both significantly reduced, were reported as markers of H. pylori infection and gastric cancer. This evidence concerns the gene PROS1 and gastric cancer.