The 315 downregulated DEGs shared among all three infection conditions enriched pathways related to ‘Cholesterol metabolism’ (Z = 16.8, P < 0.001), ‘Cholesterol biosynthesis’ (Z = 16.1, P < 0.001), ‘Integrin-mediated cell adhesion’ (Z = 4.2, P < 0.01), ‘Apoptosis’ (Z = 4.3, P < 0.01), ‘p53 signaling’ (Z = 4.2, P < 0.01), ‘SREBF and miR33 in cholesterol and lipid homeostasis’ (Z = 5.3, P < 0.01) and ‘Omega-9 FA synthesis’ (Z = 4.8, P < 0.01) (Fig. 7B). The gene discussed is TP53; the disease is infection.