The options mentioned include: monalizumab, which targets the inhibitory receptor NKG2A and stops it from suppressing the immune system; nivolumab, which blocks the interaction between the checkpoint receptor PD-1 and the PD-L1 ligand on tumor cells, thus preventing its inhibitory effects; and cetuximab, which specifically targets the EGFR receptor on melanoma cells and stops the transmission of inhibitory signals [95]. The gene discussed is KLRC1; the disease is neoplasm.