In addition to GBM, we also found that the level of CCL2 was also elevated in lymphoid neoplasm diffuse large B-cell lymphoma (DLBC), brain lower grade glioma (LGG), and uterine carcinosarcoma (UCS) (Fig. S1), which is consistent with previous reports [20–22], suggesting that CCL2 may play a role in the pathogenesis of multiple cancers including GBM. This evidence concerns the gene CCL2 and glioblastoma.