CLEC7A and inflammatory bowel disease: The nondimerizing nature of the DECTIN-1 L183F variant and its compromised role in microbial phagocytosis may explain the difficulty to clear infections with bacteria coated with DECTIN-1 ligands, and potentially contributes to the proband’s Salmonella and Clostridium infections and elevated anti-Saccharomyces (ASCA) antibody titers in the absence of other signs of inflammatory bowel disease.