For example, in hepatocellular carcinoma, miR-29c-3p suppresses cell migration, proliferation, and tumor growth via DNMT3B and LATS1-related Hippo signaling pathways, suggesting its potential as a therapeutic target7, and miR-29c-3p can get recruited by Circ-001971, thereby increasing the VEGFA level to accelerate cell proliferation, invasion, and angiogenesis of colon cancer8. The gene discussed is VEGFA; the disease is neoplasm.