By similar logic, our results show that Phox2b-expressing ventral parafacial neurons preferentially express Kcnq2 in the absence of other channel binding partners (Fig. 1, supplementary Figs. 1e and 4); therefore, we suspect this renders RTN neurons and respiratory function susceptible to Kcnq2 dysfunction, and consequently, a likely basis for breathing problems in Kcnq2 encephalopathy. The gene discussed is KCNQ2; the disease is Encephalopathy.