This result was not expected and may reflect a change in phosphorylation sites without a net change in number of phosphorylated residues of TFEB and/or heterogeneous infection rates that is not resolvable by a population-based assay such as Western blotting (58), or may suggest that TFEB is regulated through another mechanism such as the recently discovered itaconate modification of TFEB (37, 60). The gene discussed is TFEB; the disease is infection.