Patients with rhabdomyolysis commonly experience life-threatening complications, including hyperkalemia, hypercalcemia, hyperazotemia, and hyperuricemia, especially in patients with acute renal function damage.[20] Extensive muscle cell damage can cause a large influx of plasma calcium into the damaged muscles, leading to calcium phosphate precipitation and inhibition of calcitriol and parathyroid hormone resistance due to hyperphosphatemia. Here, PTH is linked to rhabdomyolysis.