CAMKK2 and Alzheimer disease: In correlation with the observation that Injection of oxytocin into the hippocampus reversed some of the damage caused by amyloid plaques in the learning and memory center in an AD animal model80, MS1262 treatment reactivated oxytocin in spinal neurons or brain signaling pathways by reversing the phosphorylation states of proteins related to synaptic plasticity and memory formation such as CAMKK1and CAMKK2 81.