This finding, along with the observation that nearly half of the AD/G9a-corregulated proteins are encoded by m6A-tagged transcripts (Fig. S4C) and the fact that AD/G9a-coregulated proteins exhibited more pronounced phosphorylation changes than expression changes, further confirmed that G9a activity broadly influences AD pathogenesis at the translational or post-translational (i.e., phosphorylation) levels. Here, EHMT2 is linked to Alzheimer disease.